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Paper Details

S-Nitrosylation of PINK1 Attenuates PINK1/Parkin-Dependent Mitophagy in hiPSC-Based Parkinson's Disease Models.
Cell Rep
90
2017
-synuclein, PARK2, PARK6, PD, PINK1, Parkin, Parkinson's Disease, Parkinson's disease, S, SNO, SNO-PINK1, cell, cell lines, hiPSC, hiPSC-, human, human-iPSC-derived neurons, mice, mitophagic dysfunction, neurodegeneration, neuronal, neuronal cell
Author NameAffiliation
Abdullah SultanScintillon Institute, USA Neuroscience and Aging Research Center, Sanford Burnham Prebys Medical Discovery Institute
Nima DolatabadiThe Scripps Research Institute, Scintillon Institute, USA Neuroscience and Aging Research Center, Sanford Burnham Prebys Medical Discovery Institute
Daniel B McClatchyThe Scripps Research Institute
John R YatesThe Scripps Research Institute
John R YatesThe Scripps Research Institute
Rajesh AmbasudhanScintillon Institute, USA Neuroscience and Aging Research Center, Sanford Burnham Prebys Medical Discovery Institute
Tomohiro NakamuraThe Scripps Research Institute, Scintillon Institute, USA Neuroscience and Aging Research Center, Sanford Burnham Prebys Medical Discovery Institute
Stuart A LiptonThe Scripps Research Institute, Scintillon Institute, USA Neuroscience and Aging Research Center, Sanford Burnham Prebys Medical Discovery Institute, University of California
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