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Paper Details

Distinct tau neuropathology and cellular profiles of an APOE3 Christchurch homozygote protected against autosomal dominant Alzheimer's dementia.
Acta Neuropathol
39
2022
ADAD, APOE, APOE3 Christchurch, APOE3 Christchurch homozygote, APOE3ch, Alzheimer's symptoms, CAA, Christchurch variant, E280A, PSEN1, PSEN1 E280A, RORB, Related Orphan Receptor B, Related Orphan Receptor B (RORB) positive neurons, autosomal dominant Alzheimer's dementia, autosomal dominant Alzheimer's disease, cerebral amyloid angiopathy, chronic inflammatory microglial profile, homeostatic astrocytes, tau
Author NameAffiliation
Daniela BoassaNational Center for Microscopy and Imaging Research (NCMIR), San Diego School of Medicine (university of california san diego), University of California
Daniela BoassaSan Diego School of Medicine (university of california san diego), University of California
Mark H EllismanNational Center for Microscopy and Imaging Research (NCMIR), San Diego School of Medicine (university of california san diego), University of California
Mark H EllismanSan Diego School of Medicine (university of california san diego), University of California
Mark H EllismanNational Center for Microscopy and Imaging Research (NCMIR), San Diego School of Medicine (university of california san diego), University of California
Mark H EllismanSan Diego School of Medicine (university of california san diego), University of California
Susanne KrasemannInstitute of Neuropathology, University Medical Center Hamburg-Eppendorf
Susanne KrasemannUniversity Medical Center Hamburg-Eppendorf
Markus GlatzelInstitute of Neuropathology, University Medical Center Hamburg-Eppendorf
Keith A JohnsonMassachusetts General Hospital, Harvard Medical School
Keith A JohnsonMassachusetts General Hospital, Harvard Medical School
Reisa A SperlingMassachusetts General Hospital, Harvard Medical School
Eric M ReimanThe Banner Alzheimer's Institute
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