Skip to Main Content

Paper Details

Integration of multiple epigenomic marks improves prediction of variant impact in saturation mutagenesis reporter assay.
Hum Mutat
30
2019
CAGI 5, base pair, cell lines, cell-types, cells, disease-associated promoters, epigenomic, epigenomic chromatin, human, noncoding regulatory element, nucleotide, participants, plasmid DNA, regulatory elements, reporter, transcription factor binding sites
Author NameAffiliation
Max SchubachBerlin Institute of Health (BIH)
Max SchubachCharite - Universitatsmedizin Berlin
Alan P BoyleUniversity of Michigan ann arbor
Alan P BoyleUniversity of Michigan ann arbor
Ivan V KulakovskiyVavilov Institute of General Genetics, Russian Academy of Sciences
Ivan V KulakovskiyLomonosov Moscow State University
Ivan V KulakovskiyInstitute of Mathematical Problems of Biology, Keldysh Institute of Applied Mathematics of Russian Academy of Sciences
Ivan V KulakovskiyEngelhardt Institute of Molecular Biology, Russian Academy of Sciences
Nir YosefDepartment of Electrical Engineering and Computer Science and Center for Computational Biology, University of California berkeley
Nir YosefDepartment of Electrical Engineering and Computer Science and Center for Computational Biology, University of California berkeley
Jay ShendureUniversity of Washington
Jay ShendureUniversity of Washington
Nadav AhituvDepartment of Bioengineering and Therapeutic Sciences and Institute for Human Genetics, University of California San Francisco
Martin KircherUniversity of Washington
Martin KircherBerlin Institute of Health (BIH)
Martin KircherCharite - Universitatsmedizin Berlin
Michael A BeerJohns Hopkins University
Michael A BeerMcKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University
  • 1 - 18

Datasets