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Paper Details

Pressure overload induces ISG15 to facilitate adverse ventricular remodeling and promote heart failure.
J Clin Invest
2
2023
DOCA, IFN, IFN-stimulated gene 15, IFNs, ISG15, ISG15 deficiency, ISGylation, Pathogenic cardiac macrophages, amino acid, angiotensin II, cardiomyocyte, cardiomyocytes, filamin-C, heart failure, humans, inflammatory cells, mice, mouse, nonischemic cardiomyopathy, rats, transverse aortic constriction, type I IFN, ubiquitin-like protein
Author NameAffiliation
Anthony O Gramolini
Anthony O GramoliniUniversity of Toronto
Anthony O Gramolini
Anthony O GramoliniUniversity of Toronto
Evgeniy V PetrotchenkoLady Davis Institute for Medical Research, Jewish General Hospital, McGill University
Christoph H BorchersLady Davis Institute for Medical Research, Jewish General Hospital, McGill University
Christoph H BorchersMcGill University
Christoph H BorchersLady Davis Institute for Medical Research, Jewish General Hospital, McGill University
Christoph H BorchersMcGill University
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