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Paper Details

SPG7 variant escapes phosphorylation-regulated processing by AFG3L2, elevates mitochondrial ROS, and is associated with multiple clinical phenotypes.
Cell Rep
29
2014
AFG3L2, ATP, Q688, SPG7, SPG7 mAAA protease, SPG7 variant, arginine, arginine 688 with glutamine, coronary artery disease, glutamine, mAAA protease, mitochondria mAAA protease, mitochondrial, oxygen, type 2 diabetes mellitus, tyrosine
Author NameAffiliation
Mary-Ellen HarperUniversity of Ottawa
Robert RobertsUniversity of Ottawa Heart Institute
Christina WillenborgUniversity of Lubeck
Jeanette ErdmannUniversity of Lubeck
Alexandre F R StewartUniversity of Ottawa Heart Institute, University of Ottawa
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