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Paper Details

Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells.
Genome Med
10
2014
CGI, CGIs, DNA, MMR deficient tumors, MMR factors, MMR genes, MMR-deficient cancers, MSH2, MSH2-deficient and -proficient human cells, MutS, MutS heterodimer subunits, Olaparib, PARP, SENP6, SUMO-protease, Saccharomyces cerevisiae, Schizosaccharomyzes pombe, Ulp2, budding, budding and fission yeast MutS, budding yeast, budding/fission yeast msh2, cancer, conserved genetic interactors, disease-relevant genes, fission, fission yeast, higher eukaryotes, human, human cells, mismatch, mismatch repair-deficient cancer, mismatch repair-deficient cancer cells, msh2, msh3, msh6, single-strand DNA breaks, tumors, yeast, yeasts
Author NameAffiliation
Elena TostiAlbert Einstein College of Medicine
Joseph A KatakowskiAlbert Einstein College of Medicine
Sonja SchaetzleinAlbert Einstein College of Medicine
Hyun-Soo KimAlbert Einstein College of Medicine
Colm J RyanUSA California Institute for Quantitative Biosciences, USA School of Medicine and Medical Science, University College Dublin
Michael Shales
Assen Roguev
Nevan J Krogan (CM4AI)USA California Institute for Quantitative Biosciences, USA J. David Gladstone Institutes
Deborah PalliserAlbert Einstein College of Medicine
Michael-Christopher KeoghAlbert Einstein College of Medicine
Winfried EdelmannAlbert Einstein College of Medicine
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