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Paper Details

BCR/ABL inhibits mismatch repair to protect from apoptosis and induce point mutations.
Cancer Res
72
2008
A, ABL, ABL-positive cells, BCR, BCR/ABL kinase-positive chronic myelogenous leukemia (CML) cells, BCR/ABL-positive cell lines, BCR/ABL-positive cells, CD34, CD34(+) CML cells, CML, DNA, EGFP, K(+), MMR, MNNG, N-methyl-N'-nitro-N-nitrosoguanidine, Na(+), Na(+)/K(+) ATPase, O(4)-methylthymine, O(6)-methylguanine, Ouabain, abl, bcr, caspase 3, chronic myelogenous leukemia (CML, enhanced green fluorescent protein, enhanced green fluorescent protein (EGFP) gene, imatinib, leukemia, leukemia cells, mutator phenotype, normal counterparts, nucleotides, p53, p73, parental cells, plasmid substrate, start codon

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