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Paper Details

Expression of a PCSK9 Gain-of-Function Mutation in C57BL/6J Mice to Facilitate Angiotensin II-Induced AAAs.
Biomolecules
2
2022
AAA, AAAs, AngII, Angiotensin II, LDL receptors, LDLR, Mice, PCSK9, Proprotein convertase subtilisin/kexin type 9, abdominal aortic aneurysms, apolipoprotein, apolipoprotein E, apolipoprotein E deficiency, cholesterol, compound deficient strains, hypercholesterolemic, low-density lipoprotein receptor, mPCSK9, mice, mouse, mouse PCSK9
Author NameAffiliation
Alan DaughertySaha Cardiovascular Research Center, College of Medicine, University of Kentucky
Alan DaughertyCollege of Medicine, University of Kentucky
Alan DaughertyCollege of Medicine, University of Kentucky
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Datasets

Release 1.2