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Paper Details

Protective and aggressive bacterial subsets and metabolites modify hepatobiliary inflammation and fibrosis in a murine model of PSC.
Gut
28
2023
BAs, PSC, RNA, SCFA, SCFAs, Vancomycin, bile acid, cholestasis, faecal 16S rRNA gene, fibrosis, hepatobiliary disease, hepatobiliary inflammation, inflammation, liver, liver disease, liver fibrosis, mice, multidrug-resistant, multidrug-resistant 2, patient, patients, primary sclerosing cholangitis, short-chain fatty acids, vancomycin
Author NameAffiliation
Jeremy WangCenter for Gastrointestinal Biology and Disease, University of North Carolina System
Jeremy WangUniversity of North Carolina System
Jeremy WangCenter for Gastrointestinal Biology and Disease, University of North Carolina System
Jeremy WangUniversity of North Carolina System
Stephanie A Montgomeryand Lineberger Comprehensive Cancer Center, University of North Carolina System
Louise B ThingholmInstitute of Clinical Molecular Biology
Andre FrankeInstitute of Clinical Molecular Biology, Christian-Albrechts-University of Kiel
Andre FrankeInstitute of Clinical Molecular Biology, Christian-Albrechts-University of Kiel
Ryan Balfour SartorUniversity of North Carolina System
Ryan Balfour SartorCenter for Gastrointestinal Biology and Disease, University of North Carolina System
Ryan Balfour SartorUniversity of North Carolina System
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