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Paper Details

Comparison of myeloid neoplasms with nonclassic 3q26.2/MECOM versus classic inv(3)/t(3;3) rearrangements reveals diverse clinicopathologic features, genetic profiles, and molecular mechanisms of MECOM activation.
Genes Chromosomes Cancer
3
2022
Author NameAffiliation
Firas H WehbeNorthwestern University Feinberg School of Medicine
Firas H WehbeNorthwestern University Feinberg School of Medicine
Lawrence J JenningsNorthwestern University Feinberg School of Medicine
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