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Paper Details

The APOE-R136S mutation protects against APOE4-driven Tau pathology, neurodegeneration and neuroinflammation.
Nat Neurosci
13
2023
AD, APOE, APOE variant, APOE3-R136S, APOE4, Alzheimer's disease, Apolipoprotein E4, LOAD, PSEN1-E280A, R136S, Single, Tau, disease-associated cell populations, human, human APOE4, human iPSC-derived neuron models, mouse, neurodegeneration, neuroinflammation, tauopathy
Author NameAffiliation
Reuben ThomasGladstone Institute of Data Science and Biotechnology, Gladstone Institutes
Eric M ReimanBanner Alzheimer's Institute
Eric M ReimanUniversity of Arizona
Robert W MahleyGladstone Institute of Neurological Disease, Gladstone Institutes
Robert W MahleyUniversity of California san francisco
Robert W MahleyUniversity of California san francisco
Robert W MahleyGladstone Institute of Neurological Disease, Gladstone Institutes
Robert W MahleyUniversity of California san francisco
Robert W MahleyUniversity of California san francisco
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