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Paper Details

SARS-CoV-2 disrupts host epigenetic regulation via histone mimicry.
Nature
53
2022
COVID-19, ORF8, ORF8 gene, SARS, SARS-, SARS-CoV, SARS-CoV-2, Severe acute respiratory syndrome coronavirus, chromatin, coronavirus disease, histone, histone H3, histone mimic site, host, host cell, host cell chromatin, host cell epigenome, human, human histone proteins, viral genome
Author NameAffiliation
Karl M GlastadEpigenetics Institute at the Perelman School of Medicine at the University of Pennsylvania
Karl M GlastadDepartment of Cell and Developmental Biology at the Perelman School of Medicine at the University of Pennsylvania
Yize LiDepartment of Microbiology at the Perelman School of Medicine at the University of Pennsylvania
Yize LiPenn Center for Research on Coronaviruses and Other Emerging Pathogens at the Perelman School of Medicine at the University of Pennsylvania
Xuping XieUniversity of Texas Medical Branch
Pei-Yong ShiUniversity of Texas Medical Branch
Benjamin A GarciaEpigenetics Institute at the Perelman School of Medicine at the University of Pennsylvania
Benjamin A GarciaDepartment of Biochemistry and Biophysics at the Perelman School of Medicine at the University of Pennsylvania
Benjamin A GarciaEpigenetics Institute at the Perelman School of Medicine at the University of Pennsylvania
Benjamin A GarciaDepartment of Biochemistry and Biophysics at the Perelman School of Medicine at the University of Pennsylvania
Shelley L BergerEpigenetics Institute at the Perelman School of Medicine at the University of Pennsylvania
Shelley L BergerDepartment of Cell and Developmental Biology at the Perelman School of Medicine at the University of Pennsylvania
Shelley L BergerDepartment of Biology at the Perelman School of Medicine at the University of Pennsylvania
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