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Paper Details

BORIS promotes chromatin regulatory interactions in treatment-resistant cancer cells.
Nature
73
2019
-, ALK, ALK-mutated, MYCN-amplified neuroblastoma10 cells, BORIS, BORIS-, CCCTC-binding factor, CTCF, CTCFL, MYCN, brother of the regulator of imprinted sites, cancer, cancer cells, cancers7, chromatin, genes, genome, loci, neuroblastoma10, oncogenes, regulatory elements1, resistant cancer cells, structural domains, super-enhancers, treatment, treatment-resistant cancer cells
Author NameAffiliation
Ruben DriesDana-Farber Cancer Institute
Ruben DriesDana-Farber Cancer Institute
Ruben DriesHarvard Medical School
Ruben DriesDana-Farber Cancer Institute
Ruben DriesHarvard Medical School
Ruben DriesDana-Farber Cancer Institute
Daniel S DayWhitehead Institute for Biomedical Research
Daniel S Day
Nathanael S GrayDana-Farber Cancer Institute
Nathanael S GrayHarvard Medical School
Nathanael S GrayDana-Farber Cancer Institute
Nathanael S GrayHarvard Medical School
Stuart H OrkinDana-Farber Cancer Institute
Stuart H OrkinBoston Children's Hospital
Stuart H OrkinHarvard Stem Cell Institute, Harvard Medical School
Stuart H OrkinHoward Hughes Medical Institute
Guo-Cheng YuanDana-Farber Cancer Institute
Guo-Cheng YuanHarvard TC Chan School of Public Health
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