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Paper Title
BORIS promotes chromatin regulatory interactions in treatment-resistant cancer cells.
PubMed
Paper Journal Title
Nature
Paper Citation Count
73
Paper Publication Year
2019
Bio Mention
-, ALK, ALK-mutated, MYCN-amplified neuroblastoma10 cells, BORIS, BORIS-, CCCTC-binding factor, CTCF, CTCFL, MYCN, brother of the regulator of imprinted sites, cancer, cancer cells, cancers7, chromatin, genes, genome, loci, neuroblastoma10, oncogenes, regulatory elements1, resistant cancer cells, structural domains, super-enhancers, treatment, treatment-resistant cancer cells
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Author Name
Affiliation
Ruben Dries
Dana-Farber Cancer Institute
Ruben Dries
Dana-Farber Cancer Institute
Ruben Dries
Harvard Medical School
Ruben Dries
Dana-Farber Cancer Institute
Ruben Dries
Harvard Medical School
Ruben Dries
Dana-Farber Cancer Institute
Daniel S Day
Whitehead Institute for Biomedical Research
Daniel S Day
Nathanael S Gray
Dana-Farber Cancer Institute
Nathanael S Gray
Harvard Medical School
Nathanael S Gray
Dana-Farber Cancer Institute
Nathanael S Gray
Harvard Medical School
Stuart H Orkin
Dana-Farber Cancer Institute
Stuart H Orkin
Boston Children's Hospital
Stuart H Orkin
Harvard Stem Cell Institute, Harvard Medical School
Stuart H Orkin
Howard Hughes Medical Institute
Guo-Cheng Yuan
Dana-Farber Cancer Institute
Guo-Cheng Yuan
Harvard TC Chan School of Public Health
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