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Paper Details

Molecular basis of ETV6-mediated predisposition to childhood acute lymphoblastic leukemia.
Blood
32
2021
-RUNX1, ALL, ETV6, ETV6 germline variants, ETV6 variants, KRAS, NRAS, PAX5, PTPN11, RUNX1, acute lymphoblastic leukemia, acute myeloid leukemia, childhood acute lymphoblastic leukemia, childhood leukemia, children, chromatin, diploid leukemia genome, germline, leukemia, leukemia genomes, leukemia predisposition syndrome, transposase, tumor, wild-type ETV6
Author NameAffiliation
Ninad OakSt. Jude Children's Research Hospital
Julie M Gastier-FosterInstitute for Genomic Medicine, Nationwide Children's Hospital
Julie M Gastier-FosterThe Ohio State University
Julie M Gastier-Foster
Julie M Gastier-FosterInstitute for Genomic Medicine, Nationwide Children's Hospital
Julie M Gastier-Foster
Julie M Gastier-FosterThe Ohio State University
Charles G Mullighan
Charles G MullighanComprehensive Cancer Center, St. Jude Children's Research Hospital
Ching-Hon PuiSt. Jude Children's Research Hospital
Ching-Hon PuiComprehensive Cancer Center, St. Jude Children's Research Hospital
Jinghui Zhang
Jinghui Zhang
Jeffery M Klco
Jeffery M KlcoComprehensive Cancer Center, St. Jude Children's Research Hospital
Stephen P HungerDepartment of Pediatrics and Center for Childhood Cancer Research, Children's Hospital of Philadelphia and the Perelman School of Medicine at The University of Pennsylvania
Stephen P HungerDepartment of Pediatrics and Center for Childhood Cancer Research, Children's Hospital of Philadelphia and the Perelman School of Medicine at The University of Pennsylvania
Mignon L LohBenioff Children's Hospital
Mignon L LohHelen Diller Family Comprehensive Cancer Center, University of California san francisco
Kim E NicholsSt. Jude Children's Research Hospital
Kim E NicholsComprehensive Cancer Center, St. Jude Children's Research Hospital
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