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Paper Details

Intracerebroventricular tempol administration in older rats reduces oxidative stress in the hypothalamus but does not change STAT3 signalling or SIRT1/AMPK pathway.
Appl Physiol Nutr Metab
8
2017
4-Hydroxy-2,2,6,6-tetramethylpiperidine-, 5'AMP, AMPK, Acetyl, Acetyl p53, Fischer 344, Hypothalamic inflammation, N-oxyl, SIRT1, STAT3, Tempol, age-related obesity, inflammation, leptin, nicotinamide adenine dinucleotide phosphate, nicotinamide adenine dinucleotide phosphate oxidase, obesity, oxidative stress, p53, rats, signal transducer and activator of transcription 3, silent mating type information regulator 2 homolog 1, tempol
AMP-Activated Protein Kinases, Aging, Animals, Cognitive Dysfunction, Crosses, Genetic, Cyclic N-Oxides, Energy Intake, Free Radical Scavengers, Hypothalamus, Infusion Pumps, Implantable, Infusions, Intraventricular, Male, Nerve Tissue Proteins, Neurons, Nootropic Agents, Obesity, Oxidative Stress, Rats, Inbred BN, Rats, Inbred F344, STAT3 Transcription Factor, Signal Transduction, Sirtuin 1, Spin Labels
Author NameAffiliation
Hale Z Toklua Geriatric Research Education and Clinical Center, Malcolm Randall Veterans Affairs Medical Center
Hale Z TokluUniversity of Florida
Philip J ScarpaceUniversity of Florida
Yasemin Kisbu-SakaryaUniversity of Florida
Nataliya Kirichenkoa Geriatric Research Education and Clinical Center, Malcolm Randall Veterans Affairs Medical Center
Nataliya KirichenkoUniversity of Florida
Michael MathenyUniversity of Florida
Erin BruceUniversity of Florida
Christy S CarterUniversity of Florida
Drake MorganUniversity of Florida
Nihal T??mera Geriatric Research Education and Clinical Center, Malcolm Randall Veterans Affairs Medical Center
Nihal T??merUniversity of Florida
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