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Paper Details

Cellular stressors contribute to the expansion of hematopoietic clones of varying leukemic potential.
Nat Commun
127
2018
DNA damage response genes, DNMT3A mutant, DNMT3A mutant clones, Hematopoietic clones, PPM1D, PPM1D mutant, PPM1D mutant clones, TP53, TP53 DNA damage response genes, hematopoietic, hematopoietic clones, long-lived clones, non-, t-AML, t-MDS
Author NameAffiliation
Christopher A MillerWashington University School of Medicine
Christopher A MillerMcDonnell Genome Institute, Washington University School of Medicine
Robert S FultonMcDonnell Genome Institute, Washington University School of Medicine
Robert S FultonMcDonnell Genome Institute, Washington University School of Medicine
Sharon E HeathWashington University School of Medicine
Peter WesterveltWashington University School of Medicine
Peter WesterveltSiteman Cancer Center, Washington University
Ravi VijWashington University School of Medicine
Ravi VijSiteman Cancer Center, Washington University
John F DiPersioWashington University School of Medicine
John F DiPersioSiteman Cancer Center, Washington University
Timothy A GraubertMassachusetts General Hospital
Matthew J WalterWashington University School of Medicine
Matthew J WalterSiteman Cancer Center, Washington University
Timothy J LeyWashington University School of Medicine
Timothy J LeySiteman Cancer Center, Washington University
Daniel C LinkWashington University School of Medicine
Daniel C LinkSiteman Cancer Center, Washington University
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