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Paper Details

Integration-independent Transgenic Huntington Disease Fragment Mouse Models Reveal Distinct Phenotypes and Life Span in Vivo.
J Biol Chem
13
2015
HD, HSP86, HSP90AA1, HTT, HTT complexes, HTT molecular complex, HTT protein, Huntington Disease, Huntington disease, Mouse, N, N171-Q148, N463-Q148, N536-Q148, N552-Q148, N586-, Q148 transgenic mice, ROSA26 locus, amino acids 171, caspase-2 fragment, caspase-6, chicken -actin promoter, cognitive decline, endogenous HTT protein, full-length HTT, human, huntingtin, mice, motor deficits, mouse, mutant, mutant HTT, mutant HTT fragments, patients, polyglutamine, psychiatric symptoms
Author NameAffiliation
Birgit SchillingFrom the Buck Institute for Research on Aging
Birgit SchillingFrom the Buck Institute for Research on Aging
Bradford W GibsonFrom the Buck Institute for Research on Aging
Bradford W GibsonFrom the Buck Institute for Research on Aging
Lisa M EllerbyFrom the Buck Institute for Research on Aging
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