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Paper Details

NF-κB inhibitor alpha has a cross-variant role during SARS-CoV-2 infection in ACE2-overexpressing human airway organoids.
bioRxiv
2
2022
ACE2, ACE2 receptor, ACE2-OE organoids, ACE2-overexpressing human, ACE2-overexpressing human airway organoids, B inhibitor alpha, NF-, NF-?, OE, OE organoids, SARS, SARS-CoV, SARS-CoV-2, adult stem cell-derived human airway organoid model, bystander cells, human, infected epithelial cells, inflammation, mRNA, primary airway cell, primary airway cell models, single
Author NameAffiliation
Camille R SimoneauGladstone Institute of Virology
Camille R SimoneauUniversity of California San Francisco
Mir M KhalidGladstone Institute of Virology
Nathan L MeyersGladstone Institute of Virology
Jennifer M HayashiGladstone Institute of Virology
Taha Y TahaGladstone Institute of Virology
Kristoffer E LeonGladstone Institute of Virology
Kristoffer E LeonUniversity of California San Francisco
Krystal A FontaineGladstone Institute of Virology
Lauren RodriguezUniversity of California San Francisco
Bastian JoehnkUniversity of California San Francisco
Michael A MatthayCardiovascular Research Institute, University of California San Francisco
Anita SilUniversity of California San Francisco
Nir YosefCenter for Computational Biology, University of California berkeley
Nir YosefWeizmann Institute of Science
Nir YosefCenter for Computational Biology, University of California berkeley
Nir YosefWeizmann Institute of Science
Melanie OttGladstone Institute of Virology
Melanie Ott
Melanie OttUniversity of California san francisco
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