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Paper Details

Signaling through FcγRIIA and the C5a-C5aR pathway mediates platelet hyperactivation in COVID-19.
bioRxiv
13
2021
C5a, C5aR, COVID-19, FcRIIA, FcRIIa, IgG, P-selectin, Patients, Platelets, SARS-CoV, SARS-CoV-2, SARS-CoV-2 infection, Syk, Thromboembolic events, cardiovascular disease, control platelets, ferritin, fostamatinib, hyperinflammation, immunothrombosis, patients, platelet, platelets, vascular complications
Author NameAffiliation
Heather M GianniniCenter for Translational Lung Biology, Lung Biology Institute, University of Pennsylvania Perelman School of Medicine
Rishi R GoelInstitute for Immunology, University of Pennsylvania Perelman School of Medicine
Ian FrankPerelman School of Medicine, University of Pennsylvania
Nuala J MeyerCenter for Translational Lung Biology, Lung Biology Institute, University of Pennsylvania Perelman School of Medicine
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