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Paper Details

MYC-driven increases in mitochondrial DNA copy number occur early and persist throughout prostatic cancer progression.
bioRxiv
1
2023
HGPIN, MYC, PCa, cancer, cancers, castration-resistant prostate cancer, cell type, high-grade prostatic intraepithelial neoplasia, luminal cells, mitochondrial, mitochondrial DNA, mouse, mtDNA, mtDNA replication genes, mtDNAcn, mtRNAs, neoplastic, neoplastic prostate cells, precancerous lesions, prostate cancer, prostate cancer cells, prostatic adenocarcinomas, prostatic cancer, tumor, tumor cell
Author NameAffiliation
Mark C MarkowskiJohns Hopkins University School of Medicine
Kenneth J PientaJohns Hopkins University School of Medicine
Kenneth J PientaJohns Hopkins University School of Medicine
Emmanuel S AntonarakisJohns Hopkins University School of Medicine
Emmanuel S AntonarakisJohns Hopkins University School of Medicine
Chi V DangJohns Hopkins University School of Medicine
Chi V DangJohns Hopkins Bloomberg School of Public Health
Chi V DangJohns Hopkins University School of Medicine
Chi V DangJohns Hopkins Bloomberg School of Public Health
Srinivasan YegnasubramanianJohns Hopkins University School of Medicine
Srinivasan YegnasubramanianJohns Hopkins University School of Medicine
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