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Paper Details

Functional-genetic dissection of HDAC dependencies in mouse lymphoid and myeloid malignancies.
Blood
46
2015
-, APL, APL cells, BCL-2, Cdkn1a, E, E-Myc, E-Myc lymphoma, G12D, HDAC, HDAC1, 2, 3, and 6, HDAC1/2, HDAC3, HDACis, HDACs, Hdac1, Hdac2, Hdac3, Hdac3-depleted cells, Hdacs, Histone deacetylase, MLL, MLL-AF9;Nras(G12D); PML-RAR acute promyelocytic leukemia [APL] cells, Nras, PML, RAR, RGFP966, Vorinostat, acute, acute promyelocytic leukemia [APL] cells, belinostat, caspase, class II and, depleted cells, hematological and solid malignancies, hematological malignancies, leukemia and lymphoma, leukemia and lymphoma cells, leukemias, lymphoid and myeloid malignancies, lymphomas, mouse, murine, myeloid leukemias, panobinostat, romidepsin, tumor
Author NameAffiliation
Eric Wang
Ricky W JohnstonePeter MacCallum Cancer Centre, University of Melbourne
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