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Paper Details

Targeted Sequencing of 10,198 Samples Confirms Abnormalities in Neuronal Activity and Implicates Voltage-Gated Sodium Channels in Schizophrenia Pathogenesis.
Biological Psychiatry
26
2019
187 genes, ARC, N-methyl-D-aspartate, N-methyl-D-aspartate receptor, NMDAR, NMDAR postsynaptic protein complexes, Schizophrenia, Sodium, activity-regulated cytoskeleton-associated protein, calcium, neuronally expressed genes, novel genes, paralog-conserved sites, schizophrenia, sodium, voltage-gated sodium channels
Author NameAffiliation
Valentina Escott-PriceDivision of Psychological Medicine and Clinical Neurosciences, Cardiff University
Derek W MorrisNational University of Ireland Galway
Michael GillDepartment of Psychiatry and Trinity Translational Medicine Institute, Trinity College Dublin
Aiden CorvinDepartment of Psychiatry and Trinity Translational Medicine Institute, Trinity College Dublin
George KirovDivision of Psychological Medicine and Clinical Neurosciences, Cardiff University
Peter HolmansDivision of Psychological Medicine and Clinical Neurosciences, Cardiff University
Michael J OwenDivision of Psychological Medicine and Clinical Neurosciences, Cardiff University
Michael C O'DonovanDivision of Psychological Medicine and Clinical Neurosciences, Cardiff University
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