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Paper Details

Genetic coding variant in complement factor B (CFB) is associated with increased risk for perianal Crohn's disease and leads to impaired CFB cleavage and phagocytosis.
Gut
4
2023
ASCA, C3b, CD, CFB, Crohn's disease, G252 CFB, G252 or, Macrophage, OmpC, Perianal Crohn's disease, Perianal complications, Recombinant S252 CFB, S252 CFB, Serine, complement factor B, human, human CFB, human CFB plasmid, macrophage, non, pCD, patients, perianal disease, recombinant, recombinant CFB, rs4151651
Author NameAffiliation
Talin HarituniansF. Widjaja Inflammatory Bowel Disease Institute, Cedars-Sinai Medical Center
Ashwin N AnanthakrishnanMassachusetts General Hospital
William A FaubionMayo Clinic
Sergio A LiraImmunology Institute, Mount Sinai Medical Center
Ryan Balfour SartorCenter for Gastrointestinal Biology and Disease, University of North Carolina
Ramnik J XavierBroad Institute Harvard
Ramnik J XavierBroad Institute Harvard
Steven R BrantRutgers Robert Wood Johnson Medical School
Judy H ChoMt Sinai School of Medicine
Richard H DuerrUniversity of Pittsburgh
John D RiouxUniversite de Montreal
Leonie SchummUniversity of Chicago
Mark S SilverbergMount Sinai Hospital, University of Toronto
Phillip FleshnerCedars-Sinai Medical Center
Gil Y MelmedF. Widjaja Inflammatory Bowel Disease Institute, Cedars-Sinai Medical Center
Christina HaF. Widjaja Inflammatory Bowel Disease Institute, Cedars-Sinai Medical Center
Gaurav SyalF. Widjaja Inflammatory Bowel Disease Institute, Cedars-Sinai Medical Center
Dermot P B McGovernF. Widjaja Inflammatory Bowel Disease Institute, Cedars-Sinai Medical Center
Dermot P B McGovernCedars-Sinai Medical Center
Dermot P B McGovernF. Widjaja Inflammatory Bowel Disease Institute, Cedars-Sinai Medical Center
Dermot P B McGovernCedars-Sinai Medical Center
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