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Paper Details

Dysregulated coordination of MAPT exon 2 and exon 10 splicing underlies different tau pathologies in PSP and AD.
Acta Neuropathol
12
2022
0N isoforms, 1N4R isoforms, AD, Alzheimer disease, MAPT, MAPT exons, MAPT exons 2 and 10, N, PSP, Tauopathies, dystrophic, dystrophic neurites, exon 10, exon 2, exons 2 and 3, human, nerurodegenerative diseases, pre, progressive supranuclear palsy, tau, tau aggregates, tau isoforms, tau neuropathologies, tau protein, tauopathies, tufted astrocytes
Author NameAffiliation
Ana C PereiraRonald M. Loeb Center for Alzheimer's Disease, Icahn School of Medicine at Mount Sinai
Ana C PereiraIcahn School of Medicine at Mount Sinai
Ana C PereiraIcahn School of Medicine at Mount Sinai
John F CraryRonald M. Loeb Center for Alzheimer's Disease, Icahn School of Medicine at Mount Sinai
John F CraryIcahn School of Medicine at Mount Sinai
John F CraryIcahn School of Medicine at Mount Sinai
Alison M GoateIcahn School of Medicine at Mount Sinai
Alison M GoateRonald M. Loeb Center for Alzheimer's Disease, Icahn School of Medicine at Mount Sinai
Alison M GoateIcahn School of Medicine at Mount Sinai
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