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Paper Details

Distinct clonal identities of B-ALLs arising after lenolidomide therapy for multiple myeloma.
Blood Adv
8
2023
B, B-ALL, B-ALL sample, B-ALL samples, B-ALLs, B-cell acute lymphoblastic leukemia, IgH, Lenolidomide, MM, Patients, TP53, TP53 variant, TP53 variants, TP53-mutant, acute myeloid leukemia, founding population, lenalidomide, malignancies, malignancy, multiple myeloma, patients, rare cells, recurrently mutated genes, tumors
Author NameAffiliation
Erica K BarnellMcDonnell Genome Institute, Washington University School of Medicine
Erica K BarnellWashington University School of Medicine
Zachary L SkidmoreMcDonnell Genome Institute, Washington University School of Medicine
Zachary L SkidmoreWashington University School of Medicine
Katie M CampbellMcDonnell Genome Institute, Washington University School of Medicine
Katie M CampbellWashington University School of Medicine
Kelsy C CottoMcDonnell Genome Institute, Washington University School of Medicine
Kelsy C CottoWashington University School of Medicine
Nicholas C SpiesMcDonnell Genome Institute, Washington University School of Medicine
Nicholas C SpiesWashington University School of Medicine
Ravi VijWashington University School of Medicine
Ravi VijSiteman Cancer Center, Washington University School of Medicine
Malachi GriffithMcDonnell Genome Institute, Washington University School of Medicine
Malachi GriffithWashington University School of Medicine
Malachi GriffithSiteman Cancer Center, Washington University School of Medicine
Malachi GriffithWashington University School of Medicine
Obi L GriffithMcDonnell Genome Institute, Washington University School of Medicine
Obi L GriffithWashington University School of Medicine
Obi L GriffithSiteman Cancer Center, Washington University School of Medicine
Obi L GriffithWashington University School of Medicine
Lukas D WartmanWashington University School of Medicine
Lukas D WartmanSiteman Cancer Center, Washington University School of Medicine
Lukas D WartmanWashington University School of Medicine
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