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Paper Details

Neuronal hyperexcitability and ion channel dysfunction in CDKL5-deficiency patient iPSC-derived cortical organoids.
Neurobiol Dis
2
2022
CDD, CDD neurons, CDKL5, CDKL5 mutation, CDKL5-deficiency, CDKL5-deficiency disorder, K<sup>+</sup>, Na<sup>+</sup>, Neuronal hyperexcitability, R59X, RTT, RTT disorders, Rett syndrome, brain organoid model, cortical organoids, epilepsy, excessive, glial cells, human, hyper-excitability, hyperexcitability, iPSC, iPSC-derived cortical organoids, neuronal, neuronal hyperexcitability, organoids, patient, patients
Author NameAffiliation
Wei WuUniversity of California San Diego
Priscilla D NegraesUniversity of California San Diego
Cleber A TrujilloUniversity of California San Diego
Alysson R MuotriUniversity of California San Diego, Center for Academic Research and Training in Anthropogeny (CARTA), Kavli Institute for Brain and Mind
Gabriel G HaddadUniversity of California San Diego, University of California San Diego School of Medicine, USA Rady Children's Hospital
Gabriel G HaddadUniversity of California San Diego, University of California San Diego School of Medicine, USA Rady Children's Hospital
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