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Paper Details

Integration of single-cell multiomic measurements across disease states with genetics identifies mechanisms of beta cell dysfunction in type 2 diabetes.
bioRxiv
2
2023
Subtype, T2D, T2D risk variants, active chromatin, beta cell, chromatin, chromatin accessibility, pancreatic islet beta cells, single, single beta cells, type 2 diabetes
Author NameAffiliation
Gaowei WangUniversity of California San Diego
Gaowei WangPediatric Diabetes Research Center, University of California San Diego
Joshua ChiouUniversity of California San Diego
Joshua ChiouUniversity of California San Diego
Joshua ChiouPediatric Diabetes Research Center, University of California San Diego
Jee Yun HanCenter for Epigenomics, University of California San Diego
Mei-Lin OkinoUniversity of California San Diego
Mei-Lin OkinoPediatric Diabetes Research Center, University of California San Diego
Seung K KimStanford University School of Medicine
Seung K KimStanford University School of Medicine
Seung K KimStanford Diabetes Research Center, Stanford University School of Medicine
Fouad Kandeel
Sebastian PreisslCenter for Epigenomics, University of California San Diego
Sebastian PreisslUniversity of Freiburg
Sebastian PreisslCenter for Epigenomics, University of California San Diego
Sebastian PreisslUniversity of Freiburg
Kyle J GaultonUniversity of California San Diego
Kyle J GaultonPediatric Diabetes Research Center, University of California San Diego
Kyle J GaultonInstitute for Genomic Medicine, University of California San Diego
Maike SanderUniversity of California San Diego
Maike SanderPediatric Diabetes Research Center, University of California San Diego
Maike SanderInstitute for Genomic Medicine, University of California San Diego
Maike SanderUniversity of California San Diego
Maike SanderMax Delbruck Center for Molecular Medicine in the Helmholtz Association
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