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Paper Details

CSF1R inhibitors exhibit antitumor activity in acute myeloid leukemia by blocking paracrine signals from support cells.
Blood
80
2019
-expressing cells, AML, AML patient samples, CSF1, CSF1R, CSF1R ligand, CSF1R-expressing cells, GW-2580, HGF, acute myeloid leukemia, colony-stimulating factor 1 (CSF1) receptor, hepatocyte growth factor, leukemia, leukemic, leukemic blasts, myeloid-lineage cells, patient, patients, primary AML, primary AML patient samples, receptor tyrosine kinase, recombinant, siRNA, small-interfering RNA, support cells, supportive cells, tyrosine, untreated cells
Anisoles, Antineoplastic Agents, Cell Differentiation, Cell Survival, Culture Media, Conditioned, Female, Humans, Leukemia, Myeloid, Acute, Male, Paracrine Communication, Prognosis, Pyrimidines, Receptors, Granulocyte-Macrophage Colony-Stimulating Factor, Survival Rate, Tumor Cells, Cultured, Tumor Microenvironment
Author NameAffiliation
David K EdwardsKnight Cancer Institute.
Kevin Watanabe-SmithKnight Cancer Institute.
Angela RofeltyKnight Cancer Institute.
Alisa DamnernsawadKnight Cancer Institute.
Ted LaderasDepartment of Medical Informatics and Clinical Epidemiology
Adam J LambleKnight Cancer Institute.
Evan F LindKnight Cancer Institute.
Andy KaempfKnight Cancer Institute, Oregon Health & Science University
Motomi MoriKnight Cancer Institute, Oregon Health & Science University
Motomi MoriSchool of Public Health, Oregon Health & Science University-Portland State University
Mara RosenbergKnight Cancer Institute.
Mara RosenbergKnight Cancer Institute.
Amanda d'AlmeidaKnight Cancer Institute.
Nicola LongKnight Cancer Institute.
Anupriya AgarwalKnight Cancer Institute.
David Tyler SweeneyKnight Cancer Institute.
Marc M LoriauxKnight Cancer Institute.
Shannon K McWeeneyDepartment of Medical Informatics and Clinical Epidemiology
Jeffrey W TynerKnight Cancer Institute.
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