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Paper Details

Mutant Kras- and p16-regulated NOX4 activation overcomes metabolic checkpoints in development of pancreatic ductal adenocarcinoma.
Nat Commun
69
2017
E2F, GAPDH, Kras, KrasG12V, Mutant Kras, NAD(P, NAD(P)H, NAD+, NADH, NF-B, NOX4, PDAC, PDAC cell, PDAC cell lines, Rb, p16, p22phox, pancreatic ductal adenocarcinoma, patient
Author NameAffiliation
Tian TianSun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine
Tian TianSun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine
Yiling LuThe University of Texas MD Anderson Cancer Center
Jun YaoThe University of Texas MD Anderson Cancer Center
Mien-Chie HungThe University of Texas MD Anderson Cancer Center
Mien-Chie HungThe University of Texas Graduate School of Biomedical Sciences
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