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Paper Details

Pathological Tau transmission initiated by binding lymphocyte-activation gene 3.
bioRxiv
2
2023
AD, Alzheimer's disease, Lag3, Tau, Tauopathies, cells, corticobasal degeneration, endogenous Tau, fibrillar form, filamentous Tau protein, lymphocyte-activation gene 3, mice, neurodegenerative diseases, neuronal Lag3, neurons, primary cortical neurons, progressive supranuclear palsy
Author NameAffiliation
Philip C WongJohns Hopkins University School of Medicine
Juan C TroncosoJohns Hopkins University School of Medicine
Juan C TroncosoJohns Hopkins University School of Medicine
Mingyao YingJohns Hopkins University School of Medicine
Mingyao YingHugo W. Moser Research Institute at Kennedy Krieger
Valina L DawsonInstitute for Cell Engineering, Johns Hopkins University School of Medicine
Valina L DawsonJohns Hopkins University School of Medicine
Valina L DawsonJohns Hopkins University School of Medicine
Valina L DawsonAdrienne Helis Malvin Medical Research Foundation
Valina L DawsonJohns Hopkins University School of Medicine
Valina L DawsonInstitute for Cell Engineering, Johns Hopkins University School of Medicine
Valina L DawsonJohns Hopkins University School of Medicine
Valina L DawsonAdrienne Helis Malvin Medical Research Foundation
Valina L DawsonJohns Hopkins University School of Medicine
Valina L DawsonJohns Hopkins University School of Medicine
Ted M DawsonInstitute for Cell Engineering, Johns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonInstitute for Cell Engineering, Johns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
Ted M DawsonJohns Hopkins University School of Medicine
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