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Paper Details

Affinity-seq detects genome-wide PRDM9 binding sites and reveals the impact of prior chromatin modifications on mammalian recombination hotspot usage.
Epigenetics Chromatin
53
2015
H3K9me2/3 histone, PRDM9, PRDM9 binding sites, PRDM9(Dom2) variant, actively transcribed genes, alleles, binding site, cognate binding sites, genes, genome-wide binding sites, genomic regions, hotspot, human, human genome, mammalian recombination hotspot, mouse, mouse genome, surrounding sequences, zinc, zinc-finger protein
Author NameAffiliation
Natalie R PowersCenter for Genome Dynamics
Matthew A HibbsCenter for Genome Dynamics
Gregory W CarterCenter for Genome Dynamics
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Release 1.2