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Paper Details

TP53 mutations and CDKN2A mutations/deletions are highly recurrent molecular alterations in the malignant progression of sinonasal papillomas.
Mod Pathol
21
2021
CCND1, CDKN2A, CDKN2A mutations, Cancer, EGFR, FGFR1, HPV, KRAS, KRAS mutations, MYC, NFE2L2, PIK3CA, PIK3CA mutations, SOX2, Sinonasal papillomas, TERT, TERT promoter, TP53, advanced sinonasal cancer, aerodigestive tract squamous cell carcinomas, benign epithelial, benign epithelial tumors, human, human papillomavirus, inverted sinonasal papillomas, low-risk human papillomavirus (HPV, oncocytic sinonasal papillomas, patients, sinonasal carcinoma, sinonasal carcinomas, sinonasal papilloma, sinonasal papilloma-, sinonasal papillomas, tumors
Author NameAffiliation
Thomas E CareyUniversity of Michigan Medical School ann arbor
Thomas E CareyUniversity of Michigan Rogel Cancer Center ann arbor
Raja R SeethalaUniversity of Pittsburgh Medical Center
Raja R SeethalaUniversity of Pittsburgh Medical Center
Scott A TomlinsUniversity of Michigan Medical School ann arbor
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Release 1.2