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Paper Details

Systematic Dissection of the Metabolic-Apoptotic Interface in AML Reveals Heme Biosynthesis to Be a Regulator of Drug Sensitivity.
Cell Metab
65
2019
ABT-199, AML, BCL-, BCL-2, BCL-2-dependent acute myeloid leukemia (AML) cell lines, Cas9, Heme, heme, metabolic genes
Author NameAffiliation
Kevin H LinDuke University School of Medicine
Abigail XieDuke University School of Medicine
Yeong-Ran AhnDuke University School of Medicine
Deborah M MuoioDuke Molecular Physiology Institute, Duke University School of Medicine
Timothy S Pardee
David A RizzieriDuke University Medical Center
Kris C WoodDuke University School of Medicine
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