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Paper Details

Rare variants implicate NMDA receptor signaling and cerebellar gene networks in risk for bipolar disorder.
Mol Psychiatry
4
2022
741 genes, Bipolar disorder, D-amino, DAO, DAO protein, Dao, N-methyl-D-aspartate, N-methyl-D-aspartate receptors, NMDA, NMDA receptor, NMDAR, NMDARs, Purkinje neurons, bipolar disorder, cerebellar gene networks, cerebellar neurons, common variant risk loci, depression, gene networks, genome sequences, granule neurons, human, human cells, mania, mental health condition, mouse, neurodevelopmental disorders, patients, risk genes
Author NameAffiliation
Tatyana ShekhtmanUniversity of California San Diego
Judith A BadnerRush University Medical College
David C GlahnBoston Children's Hospital, Harvard Medical School
David W CraigUniversity of Southern California
Howard J EdenbergIndiana University School of Medicine
Elliot S GershonUniversity of Chicago
Francis J McMahonNational Institute of Mental Health
John I NurnbergerIndiana University School of Medicine
Peter P ZandiJohns Hopkins School of Medicine
John R KelsoeUniversity of California San Diego
Jared C RoachInstitute for Systems Biology
Seth A AmentInstitute for Genome Sciences, University of Maryland School of Medicine
Seth A AmentUniversity of Maryland School of Medicine
Seth A AmentInstitute for Genome Sciences, University of Maryland School of Medicine
Seth A AmentUniversity of Maryland School of Medicine
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