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Paper Details

Loss of endogenous thymosin β<sub>4</sub> accelerates glomerular disease.
Kidney Int
24
2016
F-actin, Glomerular disease, RhoA, Thymosin 4, cultured podocytes, diabetic nephropathy, fibrosis, glomerular disease, glomerular injury, immune-mediated nephrotoxic nephritis, inflammation, mouse, nephrotoxic nephritis, periglomerular inflammation, podocyte cells, podocytes, renal, thymosin 4, unilateral ureteral obstruction
Author NameAffiliation
Maja T LindenmeyerMedical Clinic and Policlinic IV, University of Munich
Clemens D CohenMedical Clinic and Policlinic IV, University of Munich
Neil J SebireUCL Institute of Child Health
Neil J SebireUCL Institute of Child Health
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Release 1.2