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Paper Details

The E3 Ubiquitin-Protein Ligase Cullin 3 Regulates HIV-1 Transcription.
Cells
5
2020
/NFAT transcription factor binding sites, CD4+, CD4+ T cells, Cul3, Cullin 3, E3 Ubiquitin-Protein Ligase, E3-ubiquitin ligase complexes, G1, HIV-1, LTR, NF-B, NFAT, human immunodeficiency virus, human immunodeficiency virus type 1, immortalized cells, infected cells, macrophages, monocytes, primary CD4+ T cells, promotor region, target cells, ubiquitin, viral gene
Binding Sites, Blood Donors, CD4-Positive T-Lymphocytes, Cullin Proteins, Gene Expression Regulation, Viral, Gene Knockdown Techniques, HEK293 Cells, HIV Infections, HIV-1, Host-Pathogen Interactions, Humans, NF-kappa B, NFATC Transcription Factors, Terminal Repeat Sequences, Transcription, Genetic, Transfection, Virus Replication
Author NameAffiliation
Simon LangerSanford Burnham Prebys Medical Discovery Institute
Simon LangerBoehringer Ingelheim Pharma GmbH & Co. KG
Xin YinSanford Burnham Prebys Medical Discovery Institute
Arturo DiazLa Sierra University
Arturo DiazThe Nomis Center for Immunobiology and Microbial Pathogenesis, The Salk Institute for Biological Studies
Alex J PortilloSanford Burnham Prebys Medical Discovery Institute
Alex J PortilloInc.
David E GordonUniversity of California san francisco
David E GordonGladstone Institutes
David E GordonQuantitative Biosciences Institute (QBI)
Umu H RogersSanford Burnham Prebys Medical Discovery Institute
Umu H Rogersuniversity of california san diego School of Medicine, University of California
John MarlettThe Nomis Center for Immunobiology and Microbial Pathogenesis, The Salk Institute for Biological Studies
Nevan J Krogan (CM4AI)University of California san francisco
Nevan J Krogan (CM4AI)Gladstone Institutes
Nevan J Krogan (CM4AI)Quantitative Biosciences Institute (QBI)
John A T YoungRoche Pharma Research and Early Development
Lars PacheSanford Burnham Prebys Medical Discovery Institute
Sumit K ChandaSanford Burnham Prebys Medical Discovery Institute
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Release 1.2