Skip to Main Content

Paper Details

MTG16 regulates colonic epithelial differentiation, colitis, and tumorigenesis by repressing E protein transcription factors.
JCI Insight
8
2022
CAC, CBFA2T3, E protein, E protein transcription factors, Ephrussi box-binding transcription factor, IBD, MTG16, MTG16 deficiency, MTG16-deficient, Mtg16-/-, Mtg16P209T, Myeloid translocation gene 16, azoxymethane, biopsies, cancer, colitis, colitis-associated cancer, colon pathologies, colonic epithelial, enteroendocrine cell, inflammatory bowel disease, mice, mouse, murine, patients, sodium, sulfate, tumor, tumorigenesis
Author NameAffiliation
Mary Kay WashingtonVanderbilt University
Ken S LauVanderbilt University Medical Center
Ken S Lau
Ken S LauVanderbilt-Ingram Cancer Center, Vanderbilt University Medical Center
  • 1 - 4

Datasets

Release 1.2