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Paper Details

Defective fractalkine-CX3CR1 signaling aggravates neuroinflammation and affects recovery from cuprizone-induced demyelination.
J Neurochem
6
2022
CX3CR1, FKN, MS, Microglia, cuprizone, demyelinated lesions, demyelination, fractalkine, fractalkine receptor, hCX3CR1, hCX3CR1 variant, human, human polymorphic CX3CR1 (hCX3CR1 ) variant, induced gene profile, mice, microglial, microgliosis, multiple sclerosis, neuroinflammation, pathogenic microglia, remyelination, toxic gene
Author NameAffiliation
Andrew S MendiolaThe University of Texas at San Antonio
Sergio A LiraPrecision Immunology Institute Icahn School of Medicine at Mount Sinai
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Release 1.2