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Paper Details

C-terminal phosphorylation of NaV1.5 impairs FGF13-dependent regulation of channel inactivation.
J Biol Chem
22
2017
C, Ca2+, Ca2+/calmodulin-dependent protein kinase II, CaMKII, CaMKIIc, FGF13, HEK293 cells, Na+, NaV1, arrhythmias, arrhythmogenic disorders, calmodulin, failing, failing hearts, fibroblast growth factor 13, heart failure, mouse, phosphorylation sites, positions 1938
Author NameAffiliation
Matthew R Meyer
Cheryl F LichtiUniversity of Texas Medical Branch
Joan Heller BrownUniversity of California at San Diego
Reid Townsend
Reid TownsendWashington University Medical School
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