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Paper Details

Enzymatic Activity of HPGD in Treg Cells Suppresses Tconv Cells to Maintain Adipose Tissue Homeostasis and Prevent Metabolic Dysfunction.
Immunity
59
2019
15-keto PGE2, HPGD, Hpgd, Metabolic Dysfunction, Nuclear receptor peroxisome proliferator-activated receptor, PGE2, PPAR, Regulatory T cells, T cell, Tconv Cells, Treg Cells, Treg cells, VAT Treg cells, autoimmunity, humans, hydroxyprostaglandin, hydroxyprostaglandin dehydrogenase, inflammation, insulin, insulin resistance, mouse, mouse Treg cells, prostaglandin E2, type 2 diabetes
Author NameAffiliation
Kotha SubbaramaiahWeill Cornell Medical College
Susanne V SchmidtLIMES-Institute, University of Bonn
Naganari OhkuraWPI Immunology Frontier Research Center, Osaka University
Shimon SakaguchiWPI Immunology Frontier Research Center, Osaka University
Matthias BlüherUniversity of Leipzig
Andrew J DannenbergWeill Cornell Medical College
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Release 1.2