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Paper Details

Defects in muscle branched-chain amino acid oxidation contribute to impaired lipid metabolism.
Mol Metab
102
2016
BCAA, BCAA enzyme, Mut, T2D, amino acid, branched-chain amino acid, branched-chain amino acids, cultured cells, fatty acid, glucose, humans, hyperinsulinemia, insulin, insulin resistant, methylmalonyl-CoA, methylmalonyl-CoA mutase, mice, obesity, skeletal muscle, triglyceride, type 2 diabetes
Author NameAffiliation
Manway LiuBoston University
Manway LiuBoston University
Simon KasifBoston University
Simon KasifBoston University
Jonathan M DreyfussJoslin Diabetes Center, Boston University
Elvira IsganaitisJoslin Diabetes Center, USA Harvard Medical School
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