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Paper Details

SCN5A compound heterozygosity mutation in Brugada syndrome: Functional consequences and the implication for pharmacological treatment.
Life Sciences
1
2021
BrS, Brugada syndrome, G400R, Na, Na+, Nav1, Quinidine, SCN5A, SCN5A gene, SCN5A variants, SNC5A, T1461S, T1461S positions, alleles, human, human embryonic kidney (HEK293) cells, mutant Na+ channels, patient, quinidine, transfected cells
Author NameAffiliation
José Eduardo KriegerHeart Institute, University of Sao Paulo Medical School
Alexandre C PereiraHeart Institute, University of Sao Paulo Medical School
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Release 1.2