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Paper Details

KIT-Dependent and KIT-Independent Genomic Heterogeneity of Resistance in Gastrointestinal Stromal Tumors - TORC1/2 Inhibition as Salvage Strategy.
Mol Cancer Ther
21
2019
AKT, BRAF, GIST, GIST cell lines, Gastrointestinal Stromal Tumors, KIT, KIT mutations, KIT-downstream mutations, KRAS, MEK, NF1, NF1/2, PDGFRA, PIK3CA, PTEN, RAS, Sapanisertib, Sporadic gastrointestinal stromal tumors, TORC1/2, TSC1, TSC1/2, TSC2, cell lines, isogenic cell line panel, mTOR, mTOR kinase, patients, sapanisertib, tumor, tumor samples
Author NameAffiliation
Albrecht Stenzinger
Albrecht StenzingerInstitute of Pathology, Heidelberg University Hospital
Benedikt Brors
Benedikt BrorsGerman Cancer Research Center (DKFZ), Heidelberg University
Benedikt Brors
Benedikt BrorsGerman Cancer Research Center (DKFZ), Heidelberg University
Peter Horak
Peter HorakNational Center for Tumor Diseases (NCT) Heidelberg, German Cancer Research Center (DKFZ), Heidelberg University Hospital
Stefan Fr??hling
Stefan Fr??hlingNational Center for Tumor Diseases (NCT) Heidelberg, German Cancer Research Center (DKFZ), Heidelberg University Hospital
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