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Paper Details

DLX1 and the NuRD complex cooperate in enhancer decommissioning and transcriptional repression.
Development
6
2022
DLX1, Dlx1/2, NuRD, NuRD complex, OLIG2+, OLIG2+ cells, Olig1/2, Olig2, RBBP4, Rbbp4, cell, enhancer, genes, glia, neurons, progenitor, progenitor and oligodendrocyte fate, putative regulatory elements, transcription factor genes
Cell Differentiation, Genes, Homeobox, Homeodomain Proteins, Mi-2 Nucleosome Remodeling and Deacetylase Complex, Transcription Factors
Author NameAffiliation
James D PriceLangley Porter Psychiatric Institute, UCSF Weill Institute for Neurosciences, University of California San Francisco
James D PriceUniversity of California San Francisco
Susan LindtnerLangley Porter Psychiatric Institute, UCSF Weill Institute for Neurosciences, University of California San Francisco
Athena R YpsilantiLangley Porter Psychiatric Institute, UCSF Weill Institute for Neurosciences, University of California San Francisco
Fadya BinyameenLangley Porter Psychiatric Institute, UCSF Weill Institute for Neurosciences, University of California San Francisco
Jeffrey R JohnsonQuantitative Biosciences Institute, University of California San Francisco
Jeffrey R JohnsonGladstone Institute of Data Science and Biosciences, J. David Gladstone Institutes
Jeffrey R JohnsonUniversity of California San Francisco
Jeffrey R JohnsonIcahn School of Medicine at Mount Sinai
Billy W NewtonGladstone Institute of Data Science and Biosciences, J. David Gladstone Institutes
Billy W NewtonUniversity of California San Francisco
Nevan J Krogan (CM4AI)Quantitative Biosciences Institute, University of California San Francisco
Nevan J Krogan (CM4AI)Gladstone Institute of Data Science and Biosciences, J. David Gladstone Institutes
Nevan J Krogan (CM4AI)University of California San Francisco
Nevan J Krogan (CM4AI)Icahn School of Medicine at Mount Sinai
John L R RubensteinLangley Porter Psychiatric Institute, UCSF Weill Institute for Neurosciences, University of California San Francisco
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