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Paper Details

A plausibly causal functional lupus-associated risk variant in the STAT1-STAT4 locus.
Hum Mol Genet
26
2018
-STAT4 locus, B cell lines, B cells, HMG transcription factor family, HMGA1, SLE, STAT1, STAT4, Systemic lupus erythematosus, autoimmune disease, chronic, debilitating inflammation, eQTL, expression quantitative trait locus, genetic loci, genotype-, genotype-dependent repressive element, lupus, lupus disease, non, patients, risk allele, rs11889341
Author NameAffiliation
Bahram NamjouCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Bahram NamjouUniversity of Cincinnati College of Medicine
Adrienne H WilliamsCenter for Public Health Genomics and the Department of Biostatistical Sciences, Wake Forest School of Medicine
Julie T ZieglerCenter for Public Health Genomics and the Department of Biostatistical Sciences, Wake Forest School of Medicine
Barry I FreedmanWake Forest School of Medicine
Javier MartínInstituto de Parasitologia y Biomedicina Lopez-Neyra
John D ReveilleRheumatology and Clinical Immunogenetics, University of Texas Health Science Center at Houston
Kathy L MoserArthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation
Lindsey A CriswellRosalind Russell/Ephraim P Engleman Rheumatology Research Center, University of California San Francisco
Michelle PetriJohns Hopkins University School of Medicine
Deborah S Cunninghame GrahamKing's College London, Guy's Hospital
Timothy J VyseKing's College London, Guy's Hospital
Joel M GuthridgeArthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation
Joel M GuthridgeUniversity of Oklahoma Health Sciences Center
Joel M GuthridgeUniversity of Oklahoma Health Sciences Center
Patrick M GaffneyArthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation
Carl D LangefeldCenter for Public Health Genomics and the Department of Biostatistical Sciences, Wake Forest School of Medicine
Carl D LangefeldCenter for Public Health Genomics and the Department of Biostatistical Sciences, Wake Forest School of Medicine
Matthew T WeirauchCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Matthew T WeirauchCincinnati Children's Hospital Medical Center
Matthew T WeirauchUniversity of Cincinnati College of Medicine
Matthew T WeirauchCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Matthew T WeirauchUniversity of Cincinnati College of Medicine
Matthew T WeirauchCincinnati Children's Hospital Medical Center
John B HarleyCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
John B HarleyUnited States Department of Veterans Affairs Medical Center
John B HarleyUniversity of Cincinnati College of Medicine
Leah C KottyanCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Leah C KottyanUniversity of Cincinnati College of Medicine
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