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Paper Details

EVI1 phosphorylation at S436 regulates interactions with CtBP1 and DNMT3A and promotes self-renewal.
Cell Death and Disease
3
2020
AML, CDK2, CDK3 kinases, CtBP1, DNMT3A, EVI1, EVI1-, EVI1-S436A, EVI1-WT, EVI1-driven leukaemia, S436A, Wild-type EVI1, acute myeloid leukaemia, haematopoietic, haematopoietic progenitor cell, hypomethylated cell population, murine, murine haematopoietic progenitors, serine
Author NameAffiliation
Andrew J K WilliamsonUniversity of Manchester
Andrew J K WilliamsonNational Institute for Health Research Biomedical Research Centre
Anthony D WhettonUniversity of Manchester
Anthony D WhettonNational Institute for Health Research Biomedical Research Centre
Anthony D WhettonUniversity of Manchester
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Release 1.2