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Paper Details

SMARCB1 deletion in atypical teratoid rhabdoid tumors results in human endogenous retrovirus K (HML-2) expression.
Sci Rep
15
2021
AT, AT/, AT/RT, AT/RT cell lines, AT/RT cells, Atypical Teratoid Rhabdoid Tumor, CRISPR, ERK, HERV-K, HERV-K LTR, HML, HML-, HML-2, HML-2 env, HML-2 promoter, Human Endogenous Retrovirus, Human Endogenous Retrovirus K, MAPK, MYC, NRAS, NSCs, Ras, SMARCB1, atypical teratoid rhabdoid tumors, dCas9, human, human endogenous retrovirus K, human genome, neural stem cells, patient, pediatric central nervous system cancer, repetitive element, stem cell, tumor, tumor suppressor gene, undifferentiated tumors
Author NameAffiliation
Brent A OrrSt. Jude's Children's Research Hospital
Mariarita SantiPerelman School of Medicine, Children's Hospital of Philadelphia, University of Pennsylvania
Mariarita SantiPerelman School of Medicine, Children's Hospital of Philadelphia, University of Pennsylvania
Zhengping ZhuangNational Cancer Institute (NCI)
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Release 1.2