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Paper Details

A kinase-deficient NTRK2 splice variant predominates in glioma and amplifies several oncogenic signaling pathways.
Nat Commun
23
2020
Akt, NTRK, NTRK splice variants, NTRK2, NTRK2 splice variant, NTRKs, PDGF, PI3K, STAT3, T1, TrkB, TrkB.T1, Tropomyosin receptor kinase B, gene fusion, glioma, gliomas, human, neoplastic, neurotrophic tyrosine receptor kinases, novel gene fusions, splice variants, truncated NTRK2 splice, tyrosine
Author NameAffiliation
Patrick J CiminoFred Hutchinson Cancer Research Center
Patrick J CiminoUniversity of Washington School of Medicine
Frank SzulzewskyFred Hutchinson Cancer Research Center
Massimo SquatritoSeve Ballesteros Foundation Brain Tumor Group, Spanish National Cancer Research Centre
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