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Paper Details

Physiology of cardiomyocyte injury in COVID-19.
medRxiv
7
2020
COVID-19, SARS, SARS-, SARS-CoV-2, cardiac disease, cardiac dysfunction, cardiomyocyte, cardiomyocytes, heart disease, hiPSC-derived cardiomyocytes, hiPSCs, human, human cardiomyocytes, human induced pluripotent stem cells, interleukin, interleukins, myofibrils, patients, troponin, troponin-I
Author NameAffiliation
Lisa MiorinDepartment of Microbiology and Global Health and Emerging Pathogens Institute, Icahn School of Medicine at Mount Sinai
Kristin G BeaumontIcahn School of Medicine at Mount Sinai
Kris M WhiteDepartment of Microbiology and Global Health and Emerging Pathogens Institute, Icahn School of Medicine at Mount Sinai
Jens HansenDepartment of Pharmacological Sciences, and Institute for Systems Biomedicine, Icahn School of Medicine at Mount Sinai
Nicole C DuboisIcahn School of Medicine at Mount Sinai
Christoph SchanielIcahn School of Medicine at Mount Sinai
Patricia KovatchIcahn School of Medicine at Mount Sinai
Patricia KovatchIcahn School of Medicine at Mount Sinai
Joseph GoldfarbDepartment of Pharmacological Sciences, and Institute for Systems Biomedicine, Icahn School of Medicine at Mount Sinai
Mary Ann McLaughlinIcahn School of Medicine at Mount Sinai
Adolfo Garc??a-SastreDepartment of Microbiology and Global Health and Emerging Pathogens Institute, Icahn School of Medicine at Mount Sinai
Adolfo Garc??a-SastreIcahn School of Medicine at Mount Sinai
Adolfo Garc??a-SastreTisch Cancer Institute, Icahn School of Medicine at Mount Sinai
Ravi IyengarDepartment of Pharmacological Sciences, and Institute for Systems Biomedicine, Icahn School of Medicine at Mount Sinai
Ravi IyengarDepartment of Pharmacological Sciences, and Institute for Systems Biomedicine, Icahn School of Medicine at Mount Sinai
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