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Paper Details

Copy-neutral loss of heterozygosity is prevalent and a late event in the pathogenesis of FLT3/ITD AML.
Blood Cancer J
28
2014
/ITD, AML, ARs, CD33, CD34, CD34(+)/CD33(+) progenitor colonies, CD34(+)/CD33(-) progenitor colonies, CN-, FLT3, FLT3 internal tandem duplication, FLT3 locus, FLT3-activating loop mutations, FLT3/ITD, FLT3/ITD AML, FLT3/ITD-ARs, ITD, ITD AML, ITD-, ITD-ARs, LOH, Patients, acute myeloid leukemia, genes, high, leukemic, leukemic blasts, patients, wild-type FLT3
Author NameAffiliation
Derek L StirewaltClinical Research Division, Fred Hutchinson Cancer Research Center
Soheil Meshinchi1] Clinical Research Division, Fred Hutchinson Cancer Research Center, University of Washington Medical Center, Seattle Children's Hospital
Soheil Meshinchi1] Clinical Research Division, Fred Hutchinson Cancer Research Center, University of Washington Medical Center, Seattle Children's Hospital
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